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Ipsen Pharmaceuticals Ltd

Ipsen Pharmaceuticals Ltd
7 Upper Leeson Street, Dublin 4,
Telephone: +353 1 6681377
Fax: +353 1 6602818
Summary of Product Characteristics last updated on medicines.ie: 27/01/2012
SPC INCRELEX

When a pharmaceutical company changes an SPC or PIL, a new version is published on medicines.ie. For each version, we show the dates it was published on medicines.ie and the reasons for change.

Updated on 27/01/2012 and displayed until Current
Reasons for adding or updating:
  • Change to section 4.8 - Undesirable effects
  • Change to section 5.1 - Pharmacodynamic properties
  • Change to section 10 - Date of revision of the text
Date of revision of text on the SPC:   01-Jul-2010
Legal Category:   Product subject to medical prescription which may be renewed (B)

Free-text change information supplied by the pharmaceutical company

$0Major changes are required to sections      4.8$0$0$0$0$0Please note table in 5.1 need formatting  correctly, the content is correct but some rows are out of alignment and the table footnote text is not presented as such at present.$0$0Please also note section 10 requires updating.$0
Updated on 19/06/2011 and displayed until 27/01/2012
Reasons for adding or updating:
  • Change to section 4.8 - Undesirable effects
  • Change to section 5.1 - Pharmacodynamic properties
Date of revision of text on the SPC:   15-Mar-2011
Legal Category:   Product subject to medical prescription which may be renewed (B)

Free-text change information supplied by the pharmaceutical company



4.8         Undesirable effects

 

An integrated safety database from clinical studies contains 76 subjects with severe Primary IGFD treated for a mean duration of 4.4 years and representing 321 subject-years.

 

Hypoglycaemia is the most frequently reported adverse drug reaction. The thirty-six subjects (47%) who had one or more episodes of hypoglycaemia included 4 subjects who had hypoglycaemic seizure on one or more occasion. Of the 36 subjects, 12 (33%) had a history of hypoglycaemia prior to beginning treatment. The frequency of hypoglycaemia was highest in the first month of treatment, and episodes were more frequent in younger children. Symptomatic hypoglycaemia was generally avoided when a meal or snack was consumed either shortly before or after the administration of INCRELEX.

 

Injection site hypertrophy occurred in 24 subjects (32%). This reaction was generally associated with lack of proper rotation of injections. When injections were properly dispersed, the condition resolved.

 

Tonsillar hypertrophy was noted in 12 (16%) subjects, particularly in the first 1 to 2 years of therapy with lesser tonsillar growth in subsequent years.

 

Snoring, generally beginning in the first year of treatment, was reported in 17 subjects (22%).

 

Hypoacusis was reported in 15 subjects (20%).

 

Intracranial hypertension occurred in three subjects (4%). In two subjects the events resolved without interruption of INCRELEX treatment. INCRELEX treatment was discontinued in the third subject and resumed later at a lower dose without recurrence. Fourteen subjects (18%) had headache considered related to study drug.

 

During clinical trials in other indications totalling approximately 300 patients,

reports of local and/or systemic hypersensitivity were received for 8% of patients. All

cases were mild or moderate in severity and none was serious.

 

As with all protein pharmaceuticals, some patients may develop antibodies to INCRELEX. Anti-IGF‑1 antibodies were observed in 11 of 23 children with severe Primary IGFD tested during the first year of therapy. No attenuation of growth was observed as a consequence of the development of antibodies.

 

Table 1 contains very common (≥ 1/10) and common (≥ 1/100 to < 1/10) adverse reactions for which there is at least a reasonable suspicion of a causal relationship to INCRELEX treatment which occurred in clinical trials. Within each frequency grouping, undesirable effects are presented in order of decreasing seriousness.

Table 1:  Adverse Drug Reactions in Clinical Trials

System Organ Class

Adverse Reaction

Very Common

Common

Investigations

 

 

Cardiac murmur, abnormal tympanometry, abnormal echocardiogram, increased alanine aminotransferase*, increased aspartate aminotransferase*, increased weight

 

Cardiac Disorders

       

 

Cardiomegaly, ventricular hypertrophy, atrial hypertrophy*, tachycardia, tachycardia paroxysmal*, mitral valve incompetence*, tricuspid valve incompetence*

 

Congenital, Familial and Genetic Disorders

 

Congenital jaw malformation, pigmented naevus*

 

Blood and Lymphatic System Disorders

 

Thymus hypertrophy

 

Lymphadenopathy*

 

Nervous System Disorders

 

Headache

 

Convulsions, febrile convulsion*, benign intracranial hypertension, loss of consciousness*, sleep apnoea syndrome, dizziness, tremor*, restless leg syndrome*

 

Eye Disorders

 

 

Papilloedema, reduced visual acuity*, myopia*

 

Ear and Labyrinth Disorders

 

Hypoacusis

 

Otorrhoea, ear disorder*, middle ear disorder*, tympanic membrane disorder*, ear pain, ear congestion*, fluid in middle ear

 

Respiratory, Thoracic and Mediastinal Disorders

 

Tonsillar hypertrophy, snoring

 

Adenoidal hypertrophy, nasal turbinate hypertrophy*, dyspnoea*, nasal mucosal disorder*, obstructive airway disorder*, abnormal respiration*, nasal congestion, mouth breathing

 

Gastrointestinal Disorders

 

 

Vomiting, retching*, abdominal pain*, upper abdominal pain*, abdominal distension*, dysphagia*

 

Renal and Urinary Disorders

 

Nephrolithiasis*, hydronephrosis*, renal colic*

 

Skin and Subcutaneous Tissue Disorders

 

 

Skin hypertrophy, acrochordons*, abnormal hair texture*

 

Musculoskeletal and Connective Tissue Disorders

 

 

Arthralgia, pain in extremity, myalgia, scoliosis*, spinal deformity*, soft tissue disorder*, muscle cramp*, flank pain*, musculoskeletal stiffness*

 

Metabolism and Nutrition Disorders

Hypoglycaemia

Hypoglycaemic seizure, hyperglycaemia, hyperlipidaemia*, obesity*

 

Infections and Infestations

      

 

Febrile infection*, upper respiratory tract infection*, otitis media, otitis media serous, chronic otitis media serous *, otitis externa*, pharyngitis*, tonsillitis, ear infection, oral candidiasis*

 

Surgical and Medical Procedures

 

 

Adenotonsillectomy*, adenoidectomy, ear tube insertion

 

General Disorders and Administration Site Conditions

 

Injection site hypertrophy

 

Mucosal membrane hyperplasia, hypertrophy, injection site pain, injection site bruising, injection site fibrosis*, injection site reaction*, injection site swelling*, injection site induration*, injection site pigmentation changes*, mucosal oedema*, asthenia*, lethargy*, chest discomfort*

 

Reproductive System and Breast Disorders

 

 

Gynaecomastia, ovarian cyst*

Psychiatric Disorders

 

 

Depression*, sleep terror, nervousness, abnormal behaviour*, disorientation*

 

* = occurred in only 1 subject (1%)  

 

The following adverse reactions have been identified during post approval use of

INCRELEX:

-Systemic hypersensitivity: anaphylaxis, generalized urticaria, angioedema, dyspnoea.

-Local allergic reactions at the injection site: pruritus, urticaria.

 

In the post-marketing setting, the frequency of cases indicative of anaphylaxis was

estimated to be 0.3%. Symptoms included hives, angioedema, and dyspnoea, and

some patients required hospitalization. Upon re-administration, symptoms did not

re-occur in all patients.

Clinical efficacy

 

Five clinical studies (4 open-label and 1 double-blind, placebo-controlled) were conducted with INCRELEX. Subcutaneous doses of mecasermin, generally ranging from 60 to 120 mg/kg given twice daily (BID), were administered to 76 paediatric subjects with severe Primary IGFD. Patients were enrolled in the studies on the basis of extreme short stature, slow growth rates, low IGF‑1 serum concentrations, and normal GH secretion. Baseline characteristics for the patients evaluated in the primary and secondary efficacy analyses from the combined studies were (mean ± SD): chronological age (years): 6.8 ± 3.8; height (cm): 85.0 ± 15.3; height standard deviation score (SDS): -6.7 ± 1.8; height velocity (cm/yr): 2.8 ± 1.8; height velocity SDS: -3.3 ± 1.7; IGF‑1 (ng/ml): 21.9 ± 24.8; IGF‑1 SDS: ‑4.4 ± 2.0; and bone age (years): 3.9 ± 2.8. Sixty-two subjects had at least one year of treatment. Of these, 53 (85%) had Laron syndrome-like phenotype; 7 (11%) had GH gene deletion, and 1 (2%) had neutralizing antibodies to GH. Thirty-eight (61%) of the subjects were male; 49 (79%) were Caucasian. Fifty-six (90%) of the subjects were prepubertal at baseline.

 

Annual results for height velocity, height velocity SDS, and height SDS are shown in Table 2. Pre-treatment height velocity data were available for 59 subjects. The height velocities at a given year of treatment were compared by paired t-tests to the pre-treatment height velocities of the same subjects completing that treatment year.

 

Table 2:  Annual Height Results by Number of Years Treated with INCRELEX

 

Pre-Tx

Year 1

Year 2

Year 3

Year 4

Year 5

Year 6

Year 7

Year 8

Height Velocity (cm/yr)

 

 

 

 

 

 

 

 

 

N

59

59

54

48

39

21

20

16

14

Mean (SD)

2.8 (1.8)

8.0 (2.2)

5.8 (1.4)

5.5 (1.9)

4.7 (1.4)

4.7 (1.6)

4.8 (1.5)

4.6 (1.5)

4.5 (1.2)

Mean (SD) for change from pre-Tx

 

+5.2 (2.6)

+3.0 (2.3)

+2.6 (2.3)

+1.6 (2.1)

+1.5 (1.8)

+1.5 (1.7)

+1.0 (2.1)

+0.9 (2.4)

P-value for change from pre-Tx [1]

 

<0.0001

<0.0001

<0.0001

<0.0001

0.0015

0.0009

0.0897

0.2135

Height Velocity SDS

 

 

 

 

 

 

 

 

 

N

59

59

53

47

38

19

18

15

12

Mean (SD)

-3.3 (1.7)

1.9 (2.9)

-0.2 (1.6)

-0.3 (2.0)

-0.7 (1.9)

-0.6 (2.1)

-0.4 (1.4)

-0.4 (1.9)

-0.3 (1.8)

Mean (SD) for change from pre-Tx

P-value for change from pre-Tx [1]

 

+5.1 (3.1)

<0.0001

+3.2 (2.2)

<0.0001

+3.1  (2.4)

<0.0001

+2.5 (2.1)

<0.0001

+2.5 (2.2)

0.0001

+2.7 (1.7)

<0.0001

+2.5 (2.1)

0.0003

+2.8 (2.7)

0.0041

Height SDS

 

 

 

 

 

 

 

 

 

N

62

62

57

51

41

22

20

16

14

Mean (SD)

-6.7 (1.8)

-5.9 (1.7)

-5.6 (1.8)

-5.3 (1.8)

-5.3 (1.8)

-5.5 (1.8)

-5.4 (1.8)

-5.2 (2.0)

-5.2 (1.9)

Mean (SD) for change from pre-Tx

P-value for change from pre-Tx [1]

 

+0.8 (0.5)

<0.0001

+1.1 (0.8)

<0.0001

+1.4 (1.0)

<0.0001

+1.4 (1.1)

<0.0001

+1.4 (1.3)

<0.0001

+1.4 (1.2)

<0.0001

+1.4 (1.1)

0.0001

+1.6 (1.1)

<0.0001

Pre-Tx = Pre-treatment; SD = Standard Deviation; SDS = Standard Deviation Score

[1] P-values for comparison versus pre-Tx values were computed using paired t-tests.

 

Forty-seven subjects were included in an analysis of the effects of INCRELEX on bone age advancement.  The mean ± SD change in chronological age was 5.1 ± 3.0 years and the mean ± SD change in bone age was 5.8 ± 2.9 years.

 

Efficacy is dose dependent. For subjects receiving doses between 100 and 120 μg/kg BID, the mean first year height velocity was approximately 8.7 cm/yr.

 

 

Updated on 27/08/2008 and displayed until 19/06/2011
Reasons for adding or updating:
  • New SPC for new product

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Active Ingredients

 
   Mecasermin

Versions

 
27/01/2012 to Current
19/06/2011 to 27/01/2012
27/08/2008 to 19/06/2011
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Registered Address: Franklin House, 140 Pembroke Road, Dublin 4, Ireland
Registered Number: 254776
Tel: (353 1) 6603350 Fax: (353 1) 6686672 Email: info@ipha.ie

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